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Isolated neutron stars, their $γ$-ray efficiencies and EGRET observations
P. Goldoni,C. Musso
Physics , 1996,
Abstract: We examine a sample of detected and undetected Isolated Neutron Stars (INSs), selected on the basis of their energy loss and distance, in order to maximize their detection probability. Since the sample we consider encompasses more upper limits than detections, we make use, for the first time in this field of astronomy, of survival analysis procedures through the ASURV software package (Feigelson \& Nelson 1985, Isobe et al. 1986). We show that these techniques lead to a better understanding of the physical processes at work in high-energy emission from INSs. In particular, the recent detection of PSR 1951+32 and upper limits from pulsars pointed but not detected by EGRET show that the $\gamma $-ray efficiency of ISN is not correlated to any simple pulsar parameter.
Self-Regulated Learning and the Understanding of Complex Outcomes
Monique Boekaerts,Mariel Musso,Eduardo C. Cascallar
Education Research International , 2012, DOI: 10.1155/2012/686385
Abstract:
OBSTRUCTIVE NEPHROPATHY: ITS PHYSIOPATHOLOGY
Musso C,Navarro M,Jauregui R.
Electronic Journal of Biomedicine , 2011,
Abstract: Obstructive nephropathy is the functional and /or parenchymal renal damage secondary to the urinary tract occlusion at any part of it. The inducing urinary obstruction diseases can vary depending on the patient′s age and gender. There are many renal dysfunction inducing mechanisms involved in this entity: increase in the intra-luminal pressure, ureteral dilatation with ineffective ureteral peristalsis, glomerular ultrafiltration net pressure reduction, intra-renal glomerular blood flux reduction due to vasoconstriction, and local disease of chemotactic substances. Obstructive nephropathy can also lead to hypertension (vasoconstriction-hypervolemia), hyperkalemia, metabolic acidosis (aldosterone resistance), diabetes insipidus (vasopressine resistance). In conclusion, since obstructive nephropathy is a potentially reversible cause of renal dysfunction, it should always be taken into account among the differential diagnosis of renal failure inducing mechanisms.
Self-Regulated Learning and the Understanding of Complex Outcomes
Monique Boekaerts,Mariel Musso,Eduardo C. Cascallar
Education Research International , 2012, DOI: 10.1155/2012/686385
Abstract:
Which Antitrust Rules Are Suitable for Promoting Sustainable Competition in the Liner Shipping Industry?
Benacchio, M.,Ferrari, C.,Musso, E.
- , 2004,
Abstract: Hr？ak ID: 5239
ACUTE RENAL FAILURE SECONDARY TO RHABDOMYOLYSIS INDUCED BY INFLUENZA VACCINE IN AN OLD PATIENT
Musso C,Pidoux R,Mombelli C,Reynaldi J
Electronic Journal of Biomedicine , 2005,
Abstract:
PERSISTENT AND ANEMIZING GROSS HEMATURIA SOLVED WITH ENALAPRIL
Musso C,Mombelli C,Lizarraga A,Imperiali N
Electronic Journal of Biomedicine , 2005,
Abstract: ABSTRACTHematuria is the presence of an excessive number of red blood cells in the urine (at least three or more erythrocytes in a high-power field in centrifuged urine). It is categorized as microscopic when it is visible only with the aid of a microscope and macroscopic when the urine is tea-colored, pink or even red.Hematuria can result from injury to the kidney or injury to another site in the urinary tract, and renal hematuria can be caused by glomerular or non-glomerular disease.Some clinical and biochemical findings contribute to understand the origin of this problem: the presence of hematuria with clots suggests an urological cause, while the presence of red blood cell casts and/or dysmorphic red blood cells in a urine sample supports a glomerular etiology.In the present report we presented a clinical case of a patient suffering from persistent and anemizing gross hematuria secondary to a mesangial proliferative glomerulonephritis associated with thick glomerular basement membranes which was solved using enalapril.
MACROHEMATURIA PERSISTENTE Y ANEMIZANTE RESUELTA CON ENALAPRIL
Musso C,Mombelli C,Lizarraga A,Imperiali N
Electronic Journal of Biomedicine , 2005,
Abstract: ABSTRACTHematuria is the presence of an excessive number of red blood cells in the urine (at least three or more erythrocytes in a high-power field in centrifuged urine). It is categorized as microscopic when it is visible only with the aid of a microscope and macroscopic when the urine is tea-colored, pink or even red.Hematuria can result from injury to the kidney or injury to another site in the urinary tract, and renal hematuria can be caused by glomerular or non-glomerular disease.Some clinical and biochemical findings contribute to understand the origin of this problem: the presence of hematuria with clots suggests an urological cause, while the presence of red blood cell casts and/or dysmorphic red blood cells in a urine sample supports a glomerular etiology.In the present report we presented a clinical case of a patient suffering from persistent and anemizing gross hematuria secondary to a mesangial proliferative glomerulonephritis associated with thick glomerular basement membranes which was solved using enalapril.RESUMENLa hematuria es definida como la presencia de por lo menos tres o más eritrocitos por campo en una muestra de orina centrifugada, pudiéndose a su vez clasificar este problema en microhematuria: cuando sólo puede ser detectado con la ayuda del microscopio o macrohematuria cuando a simple vista se observa una orina color te, rosada o francamente roja.La hematuria puede ser producto de una lesión a nivel de la vía urinaria o a nivel renal, pudiendo ser esta última de etiología glomerular o no glomerular.Algunos datos clínicos y bioquímicos contribuyen a la comprensión acerca del origen de la hematuria: la presencia de coágulos en la orina sugiere una causa urológica, mientras que la presencia de cilindros eritrocitarios y/o eritrocitos dismórficos o acantocitos en la misma apoyan una etiología glomerular.En este reporte, presentamos un caso clínico de un paciente portador de una macrohematuria anemizante secundaria a una glomerulonefritis proliferativa mesangial asociada a la presencia de membranas basales glomerulares engrosadas, la cual se resolvió con el uso de enalapril.
Combination of oral activated charcoal plus low protein diet as a new alternative for handling in the old end-stage renal disease patients
Musso C,Michelangelo H,Reynaldi J,Martinez B
Saudi Journal of Kidney Diseases and Transplantation , 2010,
Abstract: Chronic dialysis is a valid therapeutic option in very elderly ESRD patients, even though the decision to dialyze or not has little impact on survival. Additionally, very old patients usually do not agree with starting chronic dialysis. Even though, activated charcoal is a cheap treatment for working as adsorbent for nitrogenous products its utility is very limited. We studied the combination of a low protein diet and oral activated charcoal to reduce serum urea and crea-tinine levels in very old ESRD patients who had refused to start chronic dialysis. Nine lucid, very old > 80 years, ESRD patients who had refused to start dialysis were prescribed a treatment based on a combination of a very low protein diet and oral activated charcoal (30 gram/day). None of the patients had anuria, oliguria, edema, significant metabolic acidosis or hyperkalemia. None of them had significant gastrointestinal symptoms. After one week and ten months of charcoal use signi-ficant decrease in blood urea and creatinine levels was observed and none of them required emer-gency dialysis during this time. In conclusion, in patients more than 80 years of age low protein diet and oral activated charcoal may control the uremic symptoms effectively.
Free water clearance: Its behavior in chronic renal disease at different ages
Musso C,Reynaldi J,Martinez B,Pierangelo A
Saudi Journal of Kidney Diseases and Transplantation , 2011,
Abstract:
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